Neurone Hibernation hypothesis

Neurone Hibernation hypothesis.

The hypothesis is that when stroke occurs then the nerves do not die immediately, at least in some cases but go into a sort of hibernation.

Case study

A male, 42 YO, had a stroke that paralysed his right side and also his larynx. At his wife’s request and after talking to his attending chief neurologist, who virtually gave me carte blanche saying that “his EEG shows that he is brain dead on the left side (sic. of the brain) and it would be years before he could get out of a wheelchair”, we commenced supplements. He was then given some natural medicine designed to reduce blood clots (Micelle E 1 ml bd and Meta EPA 1g tds, lecithin 2 x 3 x d) and adenosine tds (Orthoplex) and CoQ10 tds  and Brahmitone (metagenics) to improve the efficiency of his mitochondria under hypoxic conditions.

Six months later he was walking and talking normally, shook hands firmly, and reported that he “felt strength surging into my body” after taking the first dose of the supplements.

Ubiquinone (Q10) and adenosine were given in an effort to help production of mitochondrial ATP in the oxygen deprived neurones. (Henry Osiecki once gave a lecture along those lines regarding adenosine). The fact that it worked indicates that at the time of the stroke, the nerves hibernated, at least for a day or so, rather than simply necrosed immediately they became ischaemic.

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