Movement of calcium out of the bone may leave it porous that are less able to support your weight.
But it may also lead eventually to thinner bones, with the combination being even more prone to fracture. But it doesn’t stop there because also a crystalline formation of calcium in the joints may be created during this process. The sum total of these events may cause at once a weakening of bone structure, with micro-bleeds and infection accumulating in the joints, the whole event eventually causing pain. This became “crystal” clear following the brilliantly insightful hypothesis of C W Buckley. [Prof. C.W. Buckley, Uni Syd 1928: “All Arthropathy is a Result of Septic Foci”.]
Joint infection with calcification. My hypothesis is that calcium deposits sever capillaries, allowing blood to enter the joint. Resident bacteria grow well in this medium, building to an infection.
Below right depicts the calcification in the distal joints (phalanges) but also apparently also in the metacarpals and wrist.
To understand how this condition in the images above and below may be rectified, you need to know how calcium moves in and out of bones and joints.
And then by treating this aspect, recovery may be possible, no matter what the age. Having said that it is our finding that Herring’s Direction of Cure does influence recovery. That hypothesis says fundamentally that the longer you have suffered from a chronic condition then the longer it takes to heal. We currently have four MRI results showing “before and after” measurements that highlight beneficial changes accvrding to the respective diagnoses from MDs.
And having said that, we find that the judicious use of TGA-approved Frequency Specific Bioresonance (microcurrent) treatments may help to accelerate recovery from such joint conditions significantly.
Ant according to the Australian Health Survey findings of the extreme rate of calcium and other mineral deficiencies in the average Australian diet, recovery certainly does also require specific and targeted nutritional therapy.
So creating such nutritional combinations became a goal, and I like to think has helped many such conditions over 40-odd years!
Why calcium stores in bone are ever-changing
Firstly, according to the Australian Health Surveys of 2011 and 2015, it is important to understand that not all foods and water supplies contain the calcium they should.
Also that not all calcium in food is absorbed when you eat it.
And also calcium requires special biological and nutritional co-factors to enable its absorption.
Finally, calcium moves when the brain (through the pituitary gland*) tells it to. And quickly.
*Just out of interest, the pituitary gland is the third eye in Ayuvedic medicinal culture.
How calcium moves around inside your body.
Under these constantly-changing dietary conditions that constantly see changes in calcium intake, the blood level of calcium is forever changing. Because calcium is necessary also for muscle contraction and nerve impulses. bone breaks down to return stored bone calcium to the blood to maintain strict levels at times of dietary inadequacy. That’s why the brain monitoring system (pituitary gland) effects this activity, otherwise if the blood calcium level drops too low we would die.
How does it do this?
The pituitary gland does this by releasing a hormone that targets the parathyroid gland and causes it to express parathyroid hormone (PTH). The PTH in turn, indirectly stimulates the activity of osteoclasts – bone cells that are tasked specifically to break down bone. How about that? Neat, eh?
It also causes the gut to absorb more calcium via the action of vitamin D3 (cholecalciferol) and the kidneys to retain it.
Calcium build-up in joints – how come? – a question for you and me.
So, we were told by our physiology lecturers at Sydney University in 1963 that bone breaks down in the lumen of the bone. From there it is anybody’s guess because certainly nobody discussed the next step as far as I remember, and the only references that I can find are from the Clevelend and Mayo clinics that seem intent on finding mysterious medical conditions rather than simple internal responses to dietary imbalances.
So what do you reckon is happening? To me, it seems that the logical sequence is as follows:
- Widespread osteoporosis occurs in conditions of (well established) dietary deficiencies of calcium.
- The bone breaks down by reflex to release calcium because blood calcium causes muscle contraction and nerve stimulation, without which we would die.
- See the end of the article for the The Nerdy bit Phil’s hypothesis..
The above health survey reveals the shocking information that 94% of Australian schoolgirls between the age of 12 and 16 YO are deficient in calcium.
This would obviously explain the presence of scoliosis, as the statisticians who analysed the survey stated that “calcium deficiency is the number one cause of osteoporosis”.
So, the purpose of this article is to show you how to avoid potential arthropathy that is generally thought to be a genetic ageing process that is unavoidable.
Bone loss (osteoporosis) from low intake of calcium
Low intake of calcium from the diet may cause calcium to move out of bone in order to prop up lowered blood calcium levels.https://www.ncbi.nlm.nih.gov/books/NBK482128/
This leads to a condition called osteoporosis – or bone-weakening. And calcium loss is, according to The Survey, the number one reason for osteoporosis, according to the statistical analysis that it undertook.
That means that rather than developing suddenly, osteoporosis mainly happens slowly, and not because of an ageing body so much as by your diet being deficient in calcium over prolonged periods, as the abovementioned survey suggests. So if you need to be convinced of this, please consult that table.
The survey – that took 4 years to analyse, being released in 2015 following the survey period of 2010 to 2011, goes on to say that nine out of ten girls between 12 YO and 18 YO are likely to be deficient in calcium. And this deficiency by definition is quite obviously a direct cause of scoliosis.
Why? Because the rapid growth spurt at puberty onset quite obviously requires an increase in dietary intake of the elements from which the body is made. All of them! And as calcium and magnesium are the two structural elements required in thje greatest quantity – along with protein – then these requirements must be attend to.
So why has there been so much denial on the part of the entire medical/dietitian network? It is hard to say, save for the fact that nutritional availability may not form part of their undergraduate curriculum, as it didn’t form part of mine.
That came later, with independent, further studies in the field of Clinical Nutrition. Even then there was a fair bit of denial and ignorance of this matter until the results of the 2011 survey were finally analysed by the Australian Bureau of Statistics and it became shockingly evident.
Might I say that this is a direct and reprehensible flaw in the system of training medical practitioners and pharmacists – and possibly dietitians – at University level over the generations, whose students were all told that osteoporosis and scoliosis were either unknown or genetic. Full stop.
So that is what they told parents with young girls, including us.
And this apparent misinformation has led to generations of girls developing this debilitating malady into adulthood, when the bone epiphyses harden and it becomes irreversible.
And this, given that the cause has been suspected by a few of we nutritionists from the 90s or even before that, because it flew in the face of the laws of common sense and basic physiology.
So might I now state that, especially given the results of this survey, from now on, please consult a nutritionist active in this field to establish exactly how to achieve what is a disproportionately higher intake of calcium that is needed for your child. But a word of caution – please ensure that you ask them how to achieve this in balance with other minerals and other nutrient intakes. Because if you add a supplement that is incomplete or imbalanced then it may not have the desired effect – or even a damaging one.
This movement of calcium back into the blood supply from bone reserves may be – in my opinion – the ultimate cause of why calcium accumulates in cartilage, as the joints may be calcium’s main exit point from bone.
It may also accumulate on the outside of artery walls because the arteries are the first vessels to take this exodus.
But all the while, this ongoing deficiency through the years is causing osteoporosis (inadequate bone calcium and structure) according the Australian Health Survey of Usual Nutrient intakes of 2011-12 even though these details are hard to find in the most recent survey.
That is why I believe so few people were privy to this knowledge – and even then only when they self-analysed the results, as nutritionist Henry Osiecki did through those years.
Other calcium problems? Groan!
In pronounced conditions of deficiency may also plunder calcium reserves in artery and heart muscle, as opposed to the artery lumen, so creating potentially coronary chaos.
That’s because you must always have a regulated blood calcium level that is sustained between very narrow parameters by the pituitary gland. This gland constantly monitors blood calcium levels (and other elements) and organises the parathyroid glands, gut and kidneys to “give back” and prevent the excretion of calcium.
Similarly in times of excess, it will order the thyroid gland to store calcium in the bone that is excess to requirements, given sufficient absorption co-factors such as magnesium (see above reference).
These requirements are needed to allow the heart to beat, muscles to contract and many different types of nerves to work.
Known as “electrolytes”, calcium and magnesium work in tandem to allow the contraction and relaxation of these tissues respectively.
And the good part is that consequences of an imbalance in all this may not only be rectified but also prevented by harnessing these natural phenonema!
Maintaining a normal intake of sodium and potassium is also essential, as they are the other two electrolytes that keep our body “ticking”.
Artery calcification – prevention and possible reversal
But it may get worse. As the heart’s arteries are basically en route from this “flow” of concentrated calcium that has just emerged from the bones, calcium may nestle along artery walls.
In that way, calcification may eventually render the arteries brittle by building up calcium plaque, leading into middle age and ultimately cause BP to increase in later life?
I would think that that is the most likely explanation of some otherwise unexplained causes of high BP as we hit the 40s – especially if the cholesterol levels are normal.
Vitamin K2 thought to be made in the liver in small quantities is thought to be responsible for binding calcium in bones and other tissues, so it may protect arteries from undue surface calcification.
Vitamin K occurs in leafy greens and some oils such as flaxseed and walnut oils.
As it is not possible and also unethical to make prescribing suggestions without a consult, please feel free to ring and enquire. But I hasten to add that we may recommend a regime that I use and it has certainly kept my own joints stable, pain free and with minimal swelling (my D.O.B. is January 1944). Of course, the regime naturally includes a combination of choices of a balanced
and complete adjusting of your dietary essential nutrients and/or mineral deficiencies but importantly also offering a way to optimise your health while watching your weight while providing the essentials, aimed at ultimately supporting the body’s ability to remove calcification by biological chelation.
The nerdy bit – how does this work? Phil’s hypothesis.
- And given the above, known, internal sequence of events, and the known ability of joint-cartilage cells to hold calcium, the first thing that happens is that calcium emerges from osteoclast cells into capillaries within the bone crystalline matrix (calcium/protein structures).
- From there the blood in the capillaries containing the calcium flows into the joints, which explains the formation of “false bone” in joints as osteoporosis progresses.
- We were also told that this sequence of events happened when the pituitary released trophic (activating) hormones that targeted the thyroid gland to release a hormone that affected osteoblast (bone-building) cells. Again this is a logical outcome as the pituitary gland is considered to be the master gland in the brain (the Ayveduc “third eye”) that senses the presence of many different elements in the circulation that are essential to life.
- The hormones involved are calcitonin and parathyroid hormone.
- So, in summary, the calcitonin is then released by the thyroid gland under the direction of again, the pituitary (brain) gland and stimulates osteoblast (bone building) cells into action.
- The PTH is also secreted apparently by the tyyroid gland to also act on osteoblasts, but in this case to then itself release PTH to do its thing.